The Hippo Kinase LATS2 Controls Helicobacter pylori-Induced Epithelial-Mesenchymal Transition and Intestinal Metaplasia in Gastric Mucosa

 

সংরক্ষণ করুন:
গ্রন্থ-পঞ্জীর বিবরন
লেখক: Molina Castro, Silvia Elena, Tiffon, Camille, Giraud, Julie, Boeuf, Hélène, Sifré, Elodie, Giese, Alban, Belleannée, Geneviève, Lethours, Philippe, Bessède, Emilie, Mégraud, Francis, Dubus, Pierre, Staedel, Cathy, Varon, Christine
বিন্যাস: artículo original
প্রকাশনার তারিখ:2020
বিবরন:Gastric carcinoma is related mostly to CagA+-Helicobacter pylori infection, which disrupts the gastric mucosa turnover and elicits an epithelial-mesenchymal transition (EMT) and preneoplastic transdifferentiation. The tumor suppressor Hippo pathway controls stem cell homeostasis; its core, constituted by the large tumor suppressor 2 (LATS2) kinase and its substrate Yes-associated protein 1 (YAP1), was investigated in this context.
দেশ:Kérwá
প্রতিষ্ঠান:Universidad de Costa Rica
Repositorio:Kérwá
OAI Identifier:oai:kerwa.ucr.ac.cr:10669/82160
অনলাইন ব্যবহার করুন:https://www.cmghjournal.org/article/S2352-345X(19)30143-2/fulltext
https://hdl.handle.net/10669/82160
মুখ্য শব্দ:YAP1
Epithelial-to-Mesenchymal Transition
Adenocarcinoma
CagA

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